Plants detect invading pathogens using immune receptors that are located both inside the cell and at its surface. Structural and biochemical studies have transformed our understanding of how intracellular immune receptors recognise effector proteins secreted by pathogens. But far less is known about how plant cell-surface receptors recognise effectors in the apoplast – the space outside the plant cell where many fungal pathogens colonise their hosts.
This seminar will outline a predictive modelling research theme and highlight recent examples integrating physiology, phenotyping and genetics to evaluate potential interventions and guide crop adaptation.
Chloroplast-to-nucleus retrograde signaling has long been understood through transcriptional lenses, but growing evidence points to translational control as an equally decisive layer
My PhD project aims to develop and apply novel screening methodologies to improve the identification and characterisation of heat-tolerant wheat genotypes.
Climate change is quickly reshaping the environments in which wheat is cultivated, challenging global food security through increasing temperatures, heat stress, and unpredictable climatic variability.
As the key enzyme responsible for inorganic carbon uptake in most photosynthetic organisms, Rubisco exhibits poor catalytic activity and reacts promiscuously with oxygen, limiting the rate of photosynthesis. To offset this limitation, many photosynthetic organisms have evolved carbon-concentrating mechanisms (CCMs) that saturate CO2 near Rubisco, maintaining enzyme function and suppressing oxygenation.
Chloroplasts can sense environmental fluctuations via Ca2+ signaling. Environmental triggers, such as light changes, physical damage and heat waves, can induce distinct Ca2+ signatures in chloroplasts, which may help rebalance photosynthesis and stress responses under fluctuating conditions.